Well this is a surprise...................

[taxmapper]

So the neurosurgeon looked at the head scans and found nothing. 

As in nothing. 
Empty space.. 



The "menoma" thingy is smaller than a pea and he told me that I should get one more MRI in 6 months. 
If it hasn't grown, .... meh. 

If it has... 

IM DEAD!!KAPUT ITS OVER.... 

or more like, well watch it. 


Brain shrinkage... Meh... Whadda want with 56 years? 


everything else looks fine..   
Go away. 


So in 6 months well see if anything is going on. 

till then.. 

Im Abby normal.. 

[Justagirl💃]

So the neurosurgeon looked at the head scans and found nothing.

As in nothing.
Empty space..



The "menoma" thingy is smaller than a pea and he told me that I should get one more MRI in 6 months.
If it hasn't grown, .... meh.

If it has...

IM DEAD!!KAPUT ITS OVER....

or more like, well watch it.


Brain shrinkage... Meh... Whadda want with 56 years?


everything else looks fine.. 
Go away.


So in 6 months well see if anything is going on.

till then..

Im Abby normal..

Sounds like a pleasant surprise 😉

[Parity]

Sometimes just ruling some things out is good news.
 Keep up the positive attitude and humor about you.
You know we are all hoping and wishing for the best for you.  

[WPW717]

Did pituitary hormones come back normal?

[taxmapper]

Dont know yet. 

They have to run another test, and I see the Doc on Sept. 27th. 

[WPW717]

The chemo ( incomplete due to side effects ) yielded a MRI result of no more tumor. Prolactinoma. Yet the prolactin elevation remains, albeit lower than before. 
   Have spoken to other Prolactinoma patients and this is not uncommon. 
   There is a possibility that a pituitary hormone is being produced despite no solid tumor is present.
   My breasts are feeling the effects of this even before the low dose estrogen started. The increase of the patch strength still is unmeasurable on last week’s labs but the effects are noticeable. Increasing strength on the wt training and endurance is up on aerobic exercise. Happy about this.
   Hang in there, there are answers, minus individuality, the human hormonal system is incredibly complex and complicated by genetic variation.
Hoping all comes together for you soon.

[WPW717]

Great movie reference by the way.
One of my favorites and still brings me to guffawing laughter.

[taxmapper]

So, a highly unusual conversation with my Aunt last night has me thinking.
 
Perhaps I am reading too much into it, but the conversation was unusual.
First to set the stage:
My Aunt suffered a stroke early in the year and two week later lost her husband of nearly 67 years.
She is in her mid-90’s now. The middle of three sisters, she is a strong woman who now has no controllable right side. But an amazingly sharp mind and super strong will.
Her faith is also unbreakable.
 
She was brought up in a Post Victorian home with strict protocol and a place for everything and everything in its place. She does not succumb to conspiracy theories nor is overly reactionary over any discussion.
So when telling her that “things” are taking place her typical response will fall into the category of “…well that’s unusual…” “very distressing and not uplifting to discuss…”, “…perhaps there may be a more specific reason for…” and the like.
Prior to her stroke I had asked her if she knew of any unusual medical or biological fact about me. She initially told me about my foot problem. (excessively flat feet, and why I wear wedges and blocky heels).
 
But no specific answer about the rest of me.
 
The last time I talked with her about a month and a half ago, I had avoided discussions about various things said and the MRI itself. This time around she asked me about the reason why I was a bit distressed in our last conversation and I was couthed but direct that the MRI had found my head rock, and that the purpose of the MRI was to find out about my pituitary gland and the reason for my “girlymones”.
 
When I told her that I was starting to “bust out” and (as I put it, blossom) her response was (not verbatim but close) …
 
“…well its about time you find out about this as it happens to everyone…”
 

[gotgyne]

Prior to her stroke I had asked her if she knew of any unusual medical or biological fact about me. She initially told me about my foot problem. (excessively flat feet, and why I wear wedges and blocky heels).

I have pronounced flat feet too. Do you wear for this reason wedges and block heels? If yes, how do they help?

[taxmapper]

I have worn multiple versions of arch supports in the past will negative results. 

No relief of pain, and in fact caused pain because the arch is non existent, it was like walking around with a rock in my shoe. 
Additionally, many versions of arch supports would not allow me to run. 

In our house, the "hallway" between the bathroom, the downstairs to kitchen entry and bedroom, the floor sits approximately 1 inch higher than the bedroom. A sill plat at the door sits approximately 1/2 inch higher than the hallway and the floor to the bedroom is 1.5 inches lower than the top of the sill plate. 

In 2019 I was standing on the sill plate with my heels (barefoot at that moment) on the sill plate and I was rocking back and forth like an 8 year old. I nearly fell forward and put the pads of my feet on the floor essentially creating a "high heel" condition on my feet where my heels were now sitting 1.5 inches higher than the pads of my toes: to whit I stood for about 10 min. talking to the other half like that. 

That was when I realized that the heel of my foot and the pad of my foot was supporting my weight. 

There was no weight on my arch, and NO PAIN!! 

A short time later I found a pair of women's boots in my size (I have large feet) that had a wide chunky heel and looked like any boots worn by any male.   The heel is lifted by approximately 1 3/4 inches and the end result is NO PAIN!!! 

I have been buying wedge style boots and shoes ever since. 

[taxmapper]

Breast growth. And hormones. 

[gotgyne]

I have worn multiple versions of arch supports in the past will negative results.

No relief of pain, and in fact caused pain because the arch is non existent, it was like walking around with a rock in my shoe.
Additionally, many versions of arch supports would not allow me to run.

In our house, the "hallway" between the bathroom, the downstairs to kitchen entry and bedroom, the floor sits approximately 1 inch higher than the bedroom. A sill plat at the door sits approximately 1/2 inch higher than the hallway and the floor to the bedroom is 1.5 inches lower than the top of the sill plate.

In 2019 I was standing on the sill plate with my heels (barefoot at that moment) on the sill plate and I was rocking back and forth like an 8 year old. I nearly fell forward and put the pads of my feet on the floor essentially creating a "high heel" condition on my feet where my heels were now sitting 1.5 inches higher than the pads of my toes: to whit I stood for about 10 min. talking to the other half like that.

That was when I realized that the heel of my foot and the pad of my foot was supporting my weight.

There was no weight on my arch, and NO PAIN!!

A short time later I found a pair of women's boots in my size (I have large feet) that had a wide chunky heel and looked like any boots worn by any male.  The heel is lifted by approximately 1 3/4 inches and the end result is NO PAIN!!!

I have been buying wedge style boots and shoes ever since.

Perhaps I should try it too. I wear orthopedic insoles with arch support and a pad under the forefoot since early childhood. If I go only in socks I wear  elastc bandages with a pad to support my forefoot. Without them I'd be in constant pain.

[taxmapper]

More research I found these little gems: 


https://www.sciencedirect.com/topics/medicine-and-dentistry/xx-male-syndrome


46,XX male syndrome
Found in approximately 1:20,000 newborn male babies, 46,XX testicular disorder of sex development (also known as 46,XX male syndrome) is found rarely in an infertile, phenotypic man [66]. Ninety percent will have SRY (within a small distal portion of Yp) translocated to either the tip of the X chromosome (mostly an abnormal XY interchange between PRKX and PRKY) or to an autosome [67]. The remaining SRY-negative 46, XX men are presumed to have abnormalities somewhere along the genetic cascade, leading to gonadal differentiation [68]. If SRY is present in the genome, the bipotential gonads become testes, which secrete testosterone. Testosterone induces the mesonephric duct to form the distal two thirds of the epididymis, the vas deferens, and the seminal vesicles and, after conversion to dihydrotestosterone, induces the external genitalia to virilize. Mullerian-inhibiting substance, a product of Sertoli cells, induces regression of the paramesonephric duct [69]. Therefore, 46,XX men are phenotypic men in terms of internal and external anatomy, but the testes will be small, their height may be less than the average man, and the incidence of gynecomastia and cryptorchidism may be elevated [70]. However, the AZFa, AZFb, and AZFc regions are not present and spermatogenesis does not occur. In this disorder, the karyotype is prognostic and the patient does not need either testis biopsy or an attempt at TESE.
Other structural abnormalities can be found on karyotypes that are important to recognize in the infertile man [71]. Y chromosomal abnormalities specifically may include ring Y, truncated Y, isodicentric Y, and various mosaic states involving loss of the Y chromosome in a percentage of cells [34]. A ring Y results from loss of chromosomal material and circularization of the remaining material. Ring Y chromosomes are models for other structural abnormalities of the Y in that a Y chromosomal microdeletion assay must be obtained as a complementary test to determine if any of the AZF regions are present or if the replicative mishap was extensive enough that these regions are missing [72]. If all are absent, TESE will not be successful and surgery does not need to be performed. Robertsonian and reciprocal translocations occur more often in the severely oligospermic population than in the azoospermic man [73]. In these circumstances, genetic counseling is mandatory and in those in whom it is appropriate, preimplantation genetic diagnosis may increase the chances of a healthy live birth [74].
In summary, a Y chromosomal microdeletion assay and karyotype should be obtained in all NOA and severely oligospermic men before use of any ejaculated sperm or TESE. These results must be interpreted based on available knowledge to maximize the chances for, and the health of, offspring.


https://www.sciencedirect.com/topics/medicine-and-dentistry/cryptorchism


Cryptorchidism
Cryptorchidism is the failure of descent of one or both testicles into the scrotum at birth and is one of the most common urogenital birth defects seen in newborn males. Full-term boys should have both testicles in their scrotum. Boisen and coworkers [10] examined full-term Danish boys at birth and showed an incidence of cryptorchidism of 9%. However, the incidence is substantially lower in most other locations, usually reported around 3%.
It is important to recognize that because the prenatal disruption of testicular development affects both testes, cryptorchidism should be viewed as a bilateral disease, even in cases where only one testicle is absent from the scrotum. Men with a history of cryptorchidism have 4–5 times increased risk of testis cancer [11], and the risk is there in both testicles. Most men with cryptorchidism have impaired semen quality in spite of early orchidopexy [12], and the risk of reduced spermatogenesis is present in both testes highlighting the fact that cryptorchidism is a bilateral disease. Looking at the impact of this on future fertility, men with a history of one undescended testis have a lower fertility rate (number of children fathered per man) but the same paternity rate (rate of men who have fathered at least one child) as those with bilateral descended testes, while men with a history of bilateral undescended testes have both lower fertility and paternity rates [13]. There are cases of complete azoospermia among men with a history of unilateral undescended testis, and unfortunately, there are no reliable markers to predict future fertility. It is believed that early orchidopexy may be associated with some catch-up growth, and it is clear that untreated bilateral undescended testes have a poor prognosis for fertility. The reduced fertility of cryptorchid men is likely due to germ cell loss and impaired germ cell maturation as demonstrated by Hadziselimovic and coworkers [14]. This might be caused by testicular dysgenesis, and cryptorchidism is considered to be a clinical condition associated with the testicular dysgenesis syndrome.

[taxmapper]

And because of some word limit: 

https://academic.oup.com/jcem/article/92/9/3458/2597538?login=false





I am

5-10 1/2          (176 cm) 
205-210lbs      (95Kg +/-)
BMI approx. 22
Total Testosterone is at 31.1698 nmol/L  approx. 
PSA: 3.96 ng/mL
And of course the prolactin at 20.5 ng/mL. 

Undescended and now removed left testi. 

No children.

Sperm cells non existent. 

This is again something that is raising my eyebrows because there are parallels taking place here that are indicating something specific, but moreover, I cannot seem to get any doctor to pay attention. 

[taxmapper]

So this morning I put on a bra i had purchased some time back that I haven't worn much.  A black little number with a snake skin like panel pattern.  It is suppose to be "extreme" support for physical activities and the like. The band is a standard hook type but it is a racerback (I hate over the shoulder) and is a bit tight at times.  
But two things struck me this am. 

I reported back in Jan. of this year I had lost weight. I am hovering around 207-212 currently but the breasts are starting to grow again. This bra i did not fill completely when I got it, and now I do. So I know the boobs are defiantly getting bigger.  

But with the "stay in place" aspect of this bra, I felt something I did not expect. Each step forward especially on my right side, I could feel the weight of my breast pull forward. Then fall back in typical movement one would expect with full sized breasts. the sensation is new to me and I now expect more of it.  

The discovery of these (typical and normal for women) however is not so much alarming, but kinda like what happens when you learn a new aspect of something you had the entire time but didnt know what it did.